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while specific data on Gαs-biased signaling strength differences between murine and human GLP-1Rs remains limited in the literature, β-arrestin recruitment and receptor desensitization pathways play critical roles in GLP-1R function, with reduced β-arrestin recruitment associated with prolonged cAMP signaling and enhanced metabolic efficacy [21,22], and compounds that reduce β-arrestin recruitment and retain GLP-1R at the plasma membrane produce greater long-term insulin release with faster agonist dissociation rates [23]

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The needle gauge is typically 31-32, thinner than those used for blood draws